As stated in an earlier blog post their is not necessarily an inflammation stage with microfractures if no blood cells are damaged. In addition, their are fewer pain receptors in bones making it hard to feel when you've induced a microfracture. However, a fracture does create a bony callus. You can feel bony calluses on your bones that are not hidden by muscle and fat. The humerus is an example of a bone that is well hidden whereas the tibia is an example of a bone that is quite external.
Now if you can only lengthen your tibia/fibula and radius/ulna and not your femur plus humerus that would still be a notable height increase accomplishment. For the shin bones you can do sprinting(the faster the more microfractures that will be caused) and for the forearm bones you can do clap pushups on cement(the clap is not needed, only the impact). These two activities will also cause microfractures in other bones too.
Now if those two exercises worked then why aren't sprinters really tall? The lack of detectable increase in sprinters height only means that their height increase was not sufficient to be significant enough to receive attention. In order to prove that sprinting does not increase height, you have to prove that sprinters had no change in bone size as a result of their sprinting.
The fact that sprinting causes microfractures is well documented(just do a search on pubmed). If you doubt that the repeated creation and healing of microfractures can cause height gain than do a study documenting the bone size of sprinters.
If we want more height gain than sprinters and clap pushupers have achieved than we have to use more extreme measures. Also, you must cause the microfractures as a result of tensile strain. Running however may cause tensile strain in the tibia(it causes impact forces in several directions).
Actually, a good example would be the three-time gold medalist sprinter Usain Bolt: 1,96m (!) I doubt that sprinting had no effect on his bones
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ReplyDeleteSprinters do create micro fractures from the constant impact and these in turn make the bone grow. The impact influences the bone to increases in size. But the question is in what way does the bone grow. Does it grow in width or does it grow in length? Personally, I presume that the increases depend on the diet the sprinter carries. Human beings are constantly eating, By eating we decrease our HGH production. Human growth hormone is the key. The less we eat the more HGH we produce. This is why a large chunk of the taller people are mostly slender. Looking at such a big frame you would think that they eat more than shorter people, but they really don't……. For example, basketball players undergo the same micro fractures as sprinters do. But why are they taller? I'm sure it's because of the time they have their practices & games. Sprinters, sprint in the day, normally they eat right after because when they finish, it’s 5pm(Dinner). The difference with basketball players is that most of their practices & games are at night. Players come home late. Some players come home so tired that they don't even eat they just go to sleep. Sleeping after playing b ball, sprinting up and down the court I feel that this has a very powerful affect to height because HGH levels are very high. Igf1 is important, but I feel as if HGH is more or equally important. Diet wise, intermittent fasting is the way to go. Sprinting/ doing any jumping exercises to create micro fractures on an empty stomach before going to sleep is the way to increase height. I’m sure this is the best way.
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